Joshua e raizman s 25 research works with 434 citations and 2 238 reads including.
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Ns1 non structural protein is a 40 46kda highly conserved glycoprotein of the dengue virus which may play a role in virus replication enzyme linked immunoassay elisa has established that high levels of dengue ns1 antigen and up to 10ng ml of soluble hexameric ns1 are observed to be present in the serum of dengue infected patients.
Milrinone therapeutic drug monitoring in a pediatric population.
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The carboxyl groups of mua were activated for 30 min with an aqueous solution containing 0 4 mol l 1 n 3 dimethylaminopropyl n ethylcarbodiimide edc and 0 1 mol l 1 n hydroxysuccinimide nhs.
For widespread use of specific tests to be possible fast reliable and fairly simple methods are needed.
Subsequently the electrode was washed using pbs dried with.
Dengue fever is a viral disease that affects millions of people worldwide.
In infant development maternal sensitivity is a central construct in the study of the processes involved in the caregiver infant relationship and child attachment.
After sam preparation the electrode was washed in anhydrous ethanol and deionized water and dried in a flow of nitrogen gas.
The measurement of this construct that reveals its complexity through different.
The relevance of these dyadic processes for infant and later child development requires the advancing in the measurement of maternal sensitivity.
Development and validation of a quantitative.
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Infections can accelerate atherosclerosis but the mechanisms remain unresolved.
Specific tests for dengue are not usually performed due to high costs complicated procedures and in some cases long time to yield a result.
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Stroke is the leading cause of disability in adults in the uk and hemiplegic shoulder pain hsp is a common complication with a reported incidence of 6 40 hsp interferes with rehabilitation prolongs hospital stay and substantially impairs quality of life previous prospective longitudinal studies have examined hsp in patients one week or more after stroke.
Show that c pn infection induced il 1β institutes negative feedback to inhibit gpr109a abca1 expression and cholesterol efflux leading to accumulation of intracellular cholesterol.